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GLUTATHIONE AND SUPEROXIDE DISMUTASE are considered to be the most important antioxidants known to the human species. These antioxidants are vital for cellular health and protect the cell against oxygen radicals and mitochondrial oxidative stress. The status of glutathione is considered the single most accurate indicator of the health of the cell. Antioxidant levels become depleted in inflammatory and degenerative conditions and their depletions creates a vicious cycle of further oxidative stress and degeneration.
In addition to antioxidant depietion from oxidative stress, antioxidants like GSH and SOD become exhausted from exposure to everyday environmental chemicals and toxins, cigarette smoke, pharmaceutical drugs, exercise, inappropriate diet, blood sugar disorders. Trauma and alcohol intake.
Numerous health disorders and alterations in physiology have been associated with GSH and SOD depletion. GSH levels powerfully influence healthy immune functions and cellular signaling. Research has shown that T and B lymphocytes require adequate GSH for differentiation and cellular activation. Numerous studies have shown the importance of GSH levels in immunomodulatory function and its central role in a healthy functioning immune system. The importance of optimizing glutathione reserves in immune-related conditions cannot be overemphasized.
Neurodegenerative disorders have been associated with alteration in glutathione and antioxidant status. Since the tissues of the nervous system are highly oxygenated and are composed of unsaturated fatty acids, they are prone to lipid peroxidation in instances of GSH and SOD depletion. Research has shown dramatically low levels of glutathione in conditions such as Parkinson's and Alzheimer's. Since glutathione and superoxide dismutase quench the lipid peroxidation process associated with neurodegeneration, its use in chronic neurological disorders must be considered.
GSH and SOD are important substrates for hepatic detoxification. GSH is important for Phase Il conjugation, and both SOD and GSH are impoitant for Phase I oxidation/reduction reactions. When hepatic resemes of glutathione become depleted. detoxification potentials become hindered and the body becomes more susceptible to exogenous and endogenous toxins. Studies have shown that glutathione depletion contributes and is linked to liver disease, cirrhosis. hepatitis, fattv liver. And alcohol-damaced liver.
Antioxidants, particularly GSI-I and SOD. should be considered for heart health. Atherosclerosis has been associated with decreased GSH peroxidase levels, oxidative stress. and lipid peroxidation. Exogenous glutathione has demonstrated the ability to reduce lipid peroxidation, optimize eicosanoid balance, and ultimately help protect the endothelium against damage.